{
  "annotation_id": "a98b1f96-034a-459e-89d8-f96de2c19568",
  "date_created": "2026-03-02T18:01:31.987000Z",
  "date_modified": "2026-03-02T18:01:31.987000Z",
  "file_hash": "3e02b3c533f9d3899eec3e07d76ff9314a6a0807ed699b351f4a162704030e3b",
  "private": false,
  "record": {
    "abstract": "Alternative splicing allows an organism to make different proteins in\ndifferent cells at different times, all from the same gene. In a cell that uses\nalternative splicing, the total length of all the exons is much shorter than in\na cell that encodes the same set of proteins without alternative splicing. This\neconomical use of exons makes genes more stable during reproduction and\ndevelopment because a genome with a shorter exon length is more resistant to\nharmful mutations. Genomic stability may be the reason why higher vertebrates\nsplice alternatively. For a broad class of alternatively spliced genes, a\nformula is given for the increase in their stability.",
    "arxiv_id": "q-bio/0403039",
    "authors": [
      "Kevin Cahill"
    ],
    "categories": [
      "q-bio.GN",
      "q-bio.PE"
    ],
    "doi": "10.1088/1478-3967/1/2/C01",
    "journal_ref": "Physical Biology, volume 1, issue 2, pages C1 - C4, 2004",
    "title": "Alternative Splicing and Genomic Stability",
    "url": "https://arxiv.org/abs/q-bio/0403039"
  },
  "schema_id": "dorsal/arxiv",
  "source": {
    "execution_id": "a562ec9b-c472-49b2-ac5d-4ddb76f3b003",
    "id": "arXiv Dataset IDs",
    "type": "Model",
    "variant": "snapshot-2026-03-01",
    "version": "0.1.0"
  },
  "user_id": 1000002
}